quotes, trees LV Subendocardial Midmyocardial Inferolateral Hinge Point Subepicardial RV Atria Pericardium 012 Sarcoidosis Findings Linear Nodular Focal Diffuse Normal Variants Enhancing Vessels Diseases Ischemic Heart Disease Non Ischemic Myocarditis Congestive Cardiomyopathy 007 Alcoholic Congestive Cardiomyopathy Cocaine Sarcoidosis Amyloidosis Case 006 Hypertrophic Cardiomyopathy Chloroquine Cardiomyopathy Case 002 SLE Scleroderma Examples of LGE in a Variety of Nonischemic Cardiomyopathies(Top left) A 4-chamber view of patchy distribution of late midwall and epicardial late gadolinium enhancement (LGE) (arrows) in a patient with cardiac sarcoidosis. (Top right) A 3-chamber view of a midwall stripe pattern of late gadolinium enhancement (arrows) in a patient with dilated cardiomyopathy. (Middle left) A 4-chamber view of patchy epicardial and midwall late gadolinium enhancement along the lateral wall (arrows) in a patient with myocarditis. (Middle right) A midventricular short-axis image in a patient with pulmonary hypertension (HTN) with right ventricular (RV) dilation and hypertrophy (*) along with late gadolinium enhancement in the anterior and inferior right ventricular insertion points (arrows). (Bottom left) A 3-chamber view of a LGE image in a patient with cardiac amyloid. The left ventricular blood pool is nulled (*), and there is subtle circumferential subendocardial late gadolinium enhancement throughout the left ventricle. The late gadolinium enhancement is most pronounced at the base of the left ventricle within hypertrophied myocardium (arrow). (Bottom right) A midventricular short-axis image in a patient with hypertrophic cardiomyopathy with evidence of asymmetrical septal hypertrophy with extensive midwall LGE within the hypertrophied myocardium (arrows). CMP = cardiomyopathy.Patel A.R. et al Role of Cardiac Magnetic Resonance in the Diagnosis and Prognosis of Nonischemic Cardiomyopathy JACC: Cardiovascular ImagingVolume 10, Issue 10 Part A, October 2017